Rh- Negative Blood and Antediluvian Civilizations (Robert Sepehr) 818encino

https://www.youtube.com/watch?v=ls_ZtM1Itzs

Faces of Rh Negatives Today - Eye Colours
https://www.youtube.com/watch?v=cJ8pOk4INTE

This is why they are looking at DNA?

All U.S. Presidents Related by Negative Blood Types; What are the Odds?
https://www.youtube.com/watch?v=o-n5OAj8b5Y

Why are TPTB Tracking People with Rh-negative Blood so Closely?

https://www.youtube.com/watch?v=-LIEasmolww

Here we go again. WHEEEEE!

Rhesus is a type of monkey.

Rh Negative (Rh-) means rhesus negative.

I just happen to be Rh-

Therefore there can't be any MONKEYS in my ancestry (whatever ancestry that is as I'm a mutt).

Conclusion: I'm a creation of God and there is no such thing as Darwinian evolution in me since I didn't come from a monkey.

If you are Rh+ you need to figure out your own excuse for not being a Darwinian monkey's uncle.

      

Ok, throw the monkey out!

I'm  listening, post something credible on all the blood types, lets see the explanation.

I'm patient, just make it before collapse eh! Show the opposite of what i posted, I've got an open mind.
Thanks

O neg here. They sure do say some peculiar things about us.

The rhesus blood type named after the rhesus monkey was first discovered in 1937 by Karl Landsteiner and Alexander S. Wiener. The significance of the discovery was not immediately apparent and was only realized in 1940, after subsequent findings by Philip Levine and Rufus Stetson.^[1] This serum that led to the discovery was produced by immunizing rabbits with red blood cells from a rhesus macaque. The antigen that induced this immunization was designated by them as Rh factor to indicate that rhesus blood had been used for the production of the serum.<sup>[2]


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http://www.bloodjournal.org/content/bloodjournal/10/12/1241.full.pdf

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Quote :

[size=32]Rh Blood Types[/size]

Rh blood types were discovered in 1940 by Karl Landsteiner and Alexander Wiener.  This was 40 years after Landsteiner had discovered the ABO blood groups.  Over the last half century, we have learned far more about the processes responsible for Rh types.  This blood group may be the most complex genetically of all blood type systems since it involves 45 different antigens on the surface of red cells that are controlled by 2 closely linked genes on chromosome 1.

The Rh system was named after rhesus monkeys, since they were initially used in the research to make the antiserum for typing blood samples.  If the antiserum agglutinates your red cells, you are Rh+ .  If it doesn't, you are Rh- .  Despite its actual genetic complexity, the inheritance of this trait usually can be predicted by a simple conceptual model in which there are two alleles, D and d.  Individuals who are homozygous dominant (DD) or heterozygous (Dd) are Rh+.  Those who are homozygous recessive (dd) are Rh- (i.e., they do not have the key Rh antigens).

Clinically, the Rh factor, like ABO factors, can lead to serious medical complications. The greatest problem with the Rh group is not so much incompatibilities following transfusions (though they can occur) as those between a mother and her developing fetus.  Mother-fetus incompatibility occurs when the mother is Rh- (dd) and her fetus is Rh+ (DD or Dd).  Maternal antibodies can cross the placenta and destroy fetal red blood cells.  The risk increases with each pregnancy.  Europeans are the most likely to have this problem--13% of their newborn babies are at risk.  Actually only about ½ of these babies (6% of all European births) have complications.  With preventive treatment, this number can be cut down even further.  Less than 1% of those treated have trouble.  However, Rh blood type incompatibility is still the leading cause of potentially fatal blood related problems of the newborn.  In the United States, 1 out of 1000 babies are born with this condition.

Rh type mother-fetus incompatibility occurs only when an Rh+ man fathers a child with an Rh- mother.  Since an Rh+ father can have either a DD or Dd genotype, there are 2 mating combinations possible with differing risks as shown below.  Regardless of the father's genotype, if he is Rh+ and the mother is Rh-, doctors assume that there will be an incompatibility problem and act accordingly.

	Human fetus in a mother's uterus (the umbilical cord and placenta connect the fetus to its mother)

Keep in mind that only the Rh+ children (Dd) are likely to have medical complications.  When both the mother and her fetus are Rh- (dd), the birth will be normal.

The first time an Rh- woman becomes pregnant, there usually are not incompatibility difficulties for her Rh+ fetus.  However, the second and subsequent births are likely to have life-threatening problems for Rh+ fetuses.  The risk increases with each birth.  In order to understand why first born are normally safe and later children are not, it is necessary to understand some of the placenta's functions.  It is an organ that connects the fetus to the wall of the uterus via an umbilical cord.  Nutrients and the mother's antibodies regularly transfer across the placental boundary into the fetus, but her red blood cells usually do not (except in the case of an accidental rupture).  Normally, anti-Rh+ antibodies do not exist in the first-time mother unless she has previously come in contact with Rh+ blood.  Therefore, her antibodies are not likely to agglutinate the red blood cells of her Rh+ fetus.  Placental ruptures do occur normally at birth so that some fetal blood gets into the mother's system, stimulating the development of antibodies to Rh+ blood antigens.  As little as one drop of fetal blood  stimulates the production of large amounts of antibodies.  When the next pregnancy occurs, a transfer of antibodies from the mother's system once again takes place across the placental boundary into the fetus.  The anti-Rh+ antibodies that she now produces react with the fetal blood, causing many of its red cells to burst or agglutinate.  As a result, the newborn baby may have a life-threatening anemia because of a lack of oxygen in the blood.  The baby also usually is jaundiced, fevered, quite swollen, and has an enlarged liver and spleen.  This condition is called erythroblastosis fetalis .  The standard treatment in severe cases is immediate massive transfusions of Rh- blood into the baby with the simultaneous draining of the existing blood to flush out Rh+ antibodies from the mother.  This is usually done immediately following birth, but it can be done to a fetus prior to birth.  Because the baby's own Rh+ red blood cells have been replaced with Rh- ones, the mother's anti-Rh+ antibodies don't agglutinate any additional red cells.  Later, the Rh- blood will be replaced naturally as the baby gradually produces its own Rh+ blood.  Any residual anti-Rh+ antibodies from the mother will leave gradually as well because the baby does not produce them.

Erythroblastosis fetalis can be prevented for women at high risk (i.e., Rh- women with Rh+ mates or mates whose blood type is unknown) by administering a serum (Rho-GAM ) containing anti-Rh+ antibodies into the mother around the 28th week of pregnancy and again within 72 hours after the delivery of an Rh+ baby.  This must be done for the first and all subsequent pregnancies.  The injected antibodies quickly agglutinate any fetal red cells as they enter the mother's blood, thereby preventing her from forming her own antibodies.  The serum provides only a passive form of immunization and will shortly leave her blood stream.  Therefore, she does not produce any long-lasting antibodies.  This treatment can be 99% effective in preventing erythroblastosis fetalis.  Rho-GAM is also routinely given to Rh- women after a miscarriage, an ectopic pregnancy, or an induced abortion.  Without the use of Rho-GAM, an Rh- woman is likely to produce larger amounts of Rh+ antibodies every time she becomes pregnant with an Rh+ baby because she is liable to come in contact with more Rh+ blood.  Therefore, the risk of life-threatening erythroblastosis fetalis increases with each subsequent pregnancy.

Anti-Rh+ antibodies may be produced in an individual with Rh- blood as a result of receiving a mismatched blood transfusion.  When this occurs, there is likely to be production of the antibodies throughout life.  Once again, Rho-GAM can prevent this from happening.

Mother-fetus incompatibility problems can result with the ABO system also.  However, they are very rare--less than .1% of births are affected and usually the symptoms are not as severe.  It most commonly occurs when the mother is type O and her fetus is A, B, or AB.  The symptoms in newborn babies are usually jaundice, mild anemia, and elevated bilirubin levels.  These problems in a baby are usually treated successfully without blood transfusions.

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NOTE:  Identifying someone as being Rh+ or Rh- is a simplification.  There are many variations of Rh blood types depending on which of the 45 Rh antigens are present.  The most important of these antigens for mother-fetus incompatibility and transfusion problems apparently are D, C, c, E, and e.  When an individual is identified as being Rh+ or Rh-, it is usually is in reference to the D antigen.  In other words, the individual is RhD+ or RhD-.

There are other systems as mentioned... choose one... how would you find compatability, transfusion? Which one?

Point trying to get across is that these people that were - were not in Europe.

My other was as to why the Glovernment was doing all the DNA testing and what were they looking for?

Now we see everyone ? is looking at Jesus and Mary!
Form your own conclusion or what ever.

I'm done with this... no one said anyone was related to the monkey... you see that it was the serum in antigen test... just saying... if you want to add to this or correct something technical go ahead and post.

The monkey would be the only one not knowing what creation was all about.